In addition, after CYP1A1 knockdown and THC stimulation, we found that the apoptosis rate of breast cancer cells in the THC + sh-CYP1A1 group was significantly higher than that in the sh-CYP1A1 group, indicating that CYP1A1 knockdown enhances the role of THC in promoting the apoptosis of breast cancer cells. This evidence concerns the gene CYP1A1 and breast carcinoma.