A lot of cytogenetic abnormalities contributing to AML are revealed in succession, including t(8; 21), t(15; 17), and t(9; 11) chromosomal translocations, which produce fusion proteins RUNX1‐RUNX1T1, PML‐RARα, and MLL‐AF49, respectively.26 Here, RARA is linked to acute myeloid leukemia.