Interestingly, these regions are also consistent with those implicated in parvalbumin-expressing interneuron dysfunction (63–69), which is one core of schizophrenia pathophysiology, affecting neuronal synchronization and thalamocortical networks, and leading to cognitive deficits as well as hyperdopaminergia related to positive symptoms [reviewed herein (70)]. This evidence concerns the gene PVALB and Cognitive impairment.