CLTA and Alzheimer disease: For example, miR-6740–3p, which interacts with CCT2, is significantly upregulated in AD (logFC = 0.38540845, p = 2.34E-16) and miR-661, which interacts with HPCAL4, NECAP1, CLTA, and GNAI2, has been observed to be involved in AD via metabolic and stress pathways (Hojati et al., 2021).