CXCR4 and triple-A syndrome: Incoming MIF signaling received by T-cell subsets mainly originated from hModulated SMC and hFibromyocte, and MIF- (CD74+CXCR4) and MIF- (CD74+CD44) ligand–receptor pairs were more abundant across T-cell subsets in AAA than those in normal AA (Supplementary Figure S7C).