In DYT-TOR1A, seven non-manifesting and 10 manifesting patients had a common pattern of hypermetabolism in lentiform nuclei, cerebellum, and SMA, compared with normal controls, while a second pattern of hypermetabolism in the cerebellum, midbrain, and thalamus was seen in patients with lasting dystonia, compared with either non-manifesting patients, patients with dystonia in action only, or normal controls [40]. Here, TOR1A is linked to Dystonia.