This might be expected based on its role in the processing and integration of painful and non-painful somatosensory stimuli, prior studies demonstrating alterations in responses to tactile stimulation in migraine and PTH, prior evidence for atypical pain-induced secondary somatosensory cortex activation in people with migraine and ictal allodynia, and atypical sfc of secondary somatosensory cortex in those with PTH (47–50). This evidence concerns the gene PTH and migraine disorder.