By observing the gastric ICCs and their SCF/C-kit signaling pathways in FD rats, Deng found that SCF and C-kit decreased significantly in FD model rats compared with normal rats, SCF and C-kit increased in the low, medium and high dose groups of Aurantii Fructus Immaturus compared with the model group, so Aurantii Fructus Immaturus could promote the proliferation of ICCs through upregulating the expression of SCF and C-kit to promote gastric motility (Deng et al., 2018). The gene discussed is KITLG; the disease is Fabry disease.