Our results showed that serum samples from autoAbs-positive AOSD patients could more significantly inhibit IFN-γ-mediated production of IP-10 (median 24.79 pg/ml, IQR: 13.30–38.25 pg/ml) than samples from HC (104.0 pg/ml, IQR: 59.03–146.0 pg/ml, p < 0.05, Figures 3A, B). Here, CXCL10 is linked to adult-onset Still disease.