ILF3 and breast cancer: ILF3 inhibition strikingly reversed the oncogenic properties caused by upregulated circSLC38A1, including the migratory, invasive capacities and EMT phenotype (Fig. 5H–J, Fig. S4j), we also verified the biological function of ILF3 in another BC cell line, overexpression of ILF3 could effectively restore the migratory ability that reduced due to circSLC38A1 knockdown (Fig. S4k).