In addition, they showed altered expression of WT1 + KTS and ‐KTS isoforms from birth and concluded that growth restriction results in podocyte damage due to dysregulation of WT1 and that imbalance of WT1 isoforms disturbs nephrogenesis and reduces the ability to maintain podocyte integrity, rendering IUGR rats more susceptible to renal disease. This evidence concerns the gene WT1 and fetal growth restriction.