Among the 96 genes, we found 16 common genes in both datasets (BCL2, BCL2L1, C3, CCR4, CD28, CD68, CD8A, CSF1, HMOX1, IL18, IL1A, IL1B, PTGS2, PTPRC, SELP, STAT3) that were significantly altered between AL amyloidosis and MM patients (p < 0.05; Figure 2C,D). This evidence concerns the gene STAT3 and Miyoshi myopathy.