Salka et al. [24] recently reported that human infant airway epithelial cells respond to a virus mimic (double-stranded RNA) with robust production of TSLP, and in vivo, they also found that infants with higher TSLP nasal levels at admission for respiratory infections (not necessarily bronchiolitis), had an increased probability of respiratory hospitalizations or emergency room visits 12 months after discharge, suggesting a role of TSLP secretion during severe bronchiolitis in infancy and in asthma inception later in life. The gene discussed is TSLP; the disease is asthma.