To determine contributions of HLA-A or HLA-DRB1 to fucosylation-triggered anti-tumor immunity, we knocked down their C3H/HeN mouse orthologs H2K1 or EB1 (ref. 29), respectively, in SW1 tumors (Extended Data Fig. 3b) and assessed growth and itICs in vivo. The gene discussed is HLA-A; the disease is neoplasm.