IL17A and rheumatoid arthritis: An investigation launched by Chen et al. utilized NGS technologies and bioinformatics analyses to identify the expression profile of miRNAs in RA chondrocytes and to screen a series of candidate target genes.208 Overexpression of the long noncoding RNAs (lncRNAs) ZNF667-AS1, HOTAIR and MEG3 in LPS-induced chondrocytes stimulated chondrogenic proliferation and alleviated inflammation by regulating the production of inflammatory factors such as IL-6, IL-17 and IL-23, as well as the proliferation-related protein Ki67.