GABARAPL1 and legionellosis: Allograft rejection, graft-vs.-host disease, legionellosis, leishmaniasis and type I diabetes mellitus were significantly enriched in the high GABARAPL1 subgroup (Figure 8B), whereas metabolism of xenobiotics by cytochrome P450, aminoacyl-tRNA biosynthesis, butanoate metabolism, and valine, leucine and isoleucine degradation were significantly enriched in the low GABARAPL1 subgroup (Supplementary Figure 2B).