Of note, in G1/S-arrested cells, vDNA remained Golgi-associated after infection at least until 72h p.i., suggesting that it served as efficient ‘waiting room’ for HPV until cell cycle progression into mitosis and, thus, CDK1- and PLK1-mediated phosphorylations of L2 would occur (Supplementary Fig. 9). The gene discussed is PLK1; the disease is infection.