A further study found that activating compounds for NR4A2 prevent neurotoxin (6-OHDA)-induced death in primary DA neurons and rat PC12 cells, and significantly ameliorate behavioral deficits (rotation behavior toward the lesion side) in a 6-OHDA lesioned rat model of PD without detectable dyskinesia-like side effects70. The gene discussed is NR4A2; the disease is Dyskinesia.