Figure 5C showed that miR-24 could significantly weaken the transcription activities of Gli3 and Magi1. Likewise, miR-24 could also target to two fragments of HN10 (Figure 5D). Then DEF cells were additionally treated with HN10 and JDm10. The fluorescence signal driven by 3′ UTR of Gli3 was compromised by miR-24 mimics but was rescued by HN10 infection and the ectopic expression of L1 (680–709) and L3 (1218–1245) fragments of HN10 (Figure 5E), which was similar with Magi1 instead of Gli3 (Figure 5F). Here, MAGI1 is linked to infection.