CAMP and Parkinson disease: For example, a cell-penetrating artificial mitochondria-targeting peptide (CAMP) conjugated with human metalloprotein 1A (hMT1A) restored mitochondrial activity and ROS production in in vitro PD model, while injection of CAMP-hMT1A fusion protein in the brain of a mouse model of PD rescued movement impairment and dopaminergic neuronal degeneration [195].