Dong and Qu demonstrated that EBHM and its active substances (scutellarin, baicalin, and CQA) improved learning memory in animal models of Alzheimer’s disease, and the mechanism may be related to inhibition of Aβ aggregation, modulation of the cholinergic nervous system, reduction of tau protein hyperphosphorylation, resistance to oxidative stress and inflammation, and apoptosis resistance [18]. This evidence concerns the gene MAPT and Alzheimer disease.