Going one step further, clinical implications of these findings should be highlighted since genetic and endocrine dysfunctions in the complex NTF3 and its receptor (i.e., NTRK3), as well as in NTRK2 and their targets (i.e., BDNF and NTF3), have also been reported in affective disorders, attention deficit/hyperactivity disorder (ADHD), and eating disorders (EDs), among others [47,48,49,50], which are not infrequently comorbid conditions with GD [9,51]. Here, NTRK2 is linked to attention deficit-hyperactivity disorder.