IL-20 is involved in hyperplasia and inhibits keratinocyte differentiation but is also expressed in atherosclerotic plaques and has been shown to promote atherosclerosis in a mouse model [41,42]; instead, IL-16, a chemo-attractant for CD4+ T-helper cells and myeloid cells, is also expressed in atherosclerotic plaques, but may have a plaque-stabilizing effect [43,44]. Here, IL16 is linked to atherosclerosis.