Considering cell death pathways, apart from apoptosis, it is also relevant to mention an in vivo study using SAB (25–50 mg/kg) administered to Sprague Dawley rats, in which SAB was able to prevent ferroptosis in a model of myocardial infarction, confirmed through increased expression of ferroptosis biomarkers cystine/glutamate transporter (xCT), glutathione peroxidase 4, ferroportin 1 (FPN1) and ferritin heavy chain (FTH1) when compared to animals with myocardial infarction [115]. This evidence concerns the gene FTH1 and myocardial infarction.