YAP1 and myocardial infarction: The deletion of the YAP/TAZ in cardiac fibroblasts ameliorates adverse cardiac remodeling after an MI by regulating the fibroblast proliferation and transdifferentiation into myofibroblasts and by regulating the cardiac fibroblast activation and fibroinflammatory responses downstream of the Wnt and TGF-β signaling pathways [67].