IL1B and myeloid sarcoma: Of note, the significantly increased levels of TNFα/IL1β in MS/EAE, lead to an increased surface expression of the Ca2+-permeable AMPA receptors that lack the GluA2 subunit and thus lead to an enhancement of excitatory synaptic signaling [85,129,142,162,163,173,292,293,294,392,393].