Because of the evidence supporting that EBV infection influences the development of gastric lesions in cooperation with H. pylori, and that H. pylori affects hepcidin levels and iron concentration through erosion of the gastric mucosa [14,21,22,23,33,35,36,37,38,39,40,41], we decided to evaluate the expression of HAMP (encodes for hepcidin), A1BG (encodes for AGP), and CRP (encodes for CRP) in the transcriptomic and genomic database of gastric cancer of the TCGA consortium (N = 250). The gene discussed is CRP; the disease is Epstein-Barr virus infection.