Next to this, C/EBP-members are often similarly regulated, as has been shown, for instance, during the acute phase response for IL-1b-inducible C/EBPβ and C/EBPδ [22] and in AML for 1α,25-dihydroxyvitamin-induced C/EBPβ and C/EBPδ, as well as all-trans-retinoic acid-induced C/EBPβ and C/EBPε [23], further implying a synergistic behavior and potential redundancy of the C/EBP-family members. Here, CEBPA is linked to acute myeloid leukemia.