The greater uniformity of autoimmune symptoms in MIS-C and KD was reflected in a greater synchrony of TCR mimics of human proteins, collagens, myosins and glutamate receptors all being possible targets of smooth and cardiac muscle autoimmunity [66,69,70,71,102] and phosphatases being possible targets in anti-phospholipid syndrome (APS). The gene discussed is MYH14; the disease is autoimmune polyendocrinopathy.