Given recent evidence suggesting that Vwf-deficient mice, ref. [42] as well as mice lacking the extracellular domain of GPIbα exhibited diminished leukocyte recruitment following stenosis of IVC and were protected against DVT, ref. [10] it can be inferred that an appropriate proportion of leukocyte capture at sites of DVT occurs in a VWF- and platelet-dependent manner. The gene discussed is GP1BA; the disease is deep vein thrombosis.