IL4 and autoimmune myocarditis: Thus, the blockade of IL-4, one of the key effector cytokines of Th2 in mice with experimental autoimmune myocarditis, accompanied by a decrease in the severity of the disease that was expressed in a decrease in the proportion of Th2 and an increase in the proportion of Th1 cells, reduction in vitro production IL-4, IL-5 and IL-13 against the background of increased synthesis and secretion of IFNγ [96].