As shown in Figure 1, by exogenously expressing SNAI2 in SiHa cells, SiHa-SNAI2 cells exhibited inhibited cell growth, decreased tumorsphere formation, enhanced sensitivity to cisplatin, reduced stem-cell-related factor expression, a slower tumor growth rate, and a lower tumor initiating frequency, indicating that SNAI2 plays an inhibitory role in regulating a stem-like phenotype in cervical cancer cells. The gene discussed is SNAI2; the disease is neoplasm.