SNAI2 and neoplasm: Therefore, the tumor formation assays revealed that the xenograft tumors derived from SiHa-SNAI2 cells grew much slower, and had a lower tumor initiating frequency, higher tumor-free rate, and longer tumor latency, compared with SiHa-Vec cells, suggesting that the tumorigenic capacity was attenuated by exogenous expression of SNAI2 in cervical cancer cells in vivo.