In our network, FABP physically and functionally interacts with HNF-4α, which in turn directly regulates the majority of the other proteins up-regulated in LC-IPF, such as TTHY, ALBU, RET4, and FETUA; the last two are also known as hepatokines linked to the induction of metabolic dysfunctions [48]. The gene discussed is AHSG; the disease is laryngotracheoesophageal cleft.