Furthermore, for apolipoprotein C-III the total levels were reported to be dysregulated in relation to COVID-19, confirming the observed changes in the degradation pattern with an increase of cutting efficiency at position 28 that justifies the increase of S21EAEDASL28 peptide, and the concomitant decrease of 21–84 and 21–34 fragments upon infection [41,42]. Here, APOC3 is linked to COVID-19.