This approach enhanced a response per oocytes (8–14 oocytes) (43.3% vs. 38.4%), fewer poor/excessive responses (<4 oocytes when AMH < 15 pmol/L—11.8% vs. 17.9%)/(≥15 or ≥20 oocytes when AMH ≥ 15 pmol/L—27.9% vs. 35.1%, 10.1% vs. 15.6%), and fewer actions carried out to prevent OHSS (2.3% vs. 4.5%), despite oocyte yielding, blastocysts number, and gonadotropin use (10.0% vs. 10.4%, 3.3% vs. 3.5%, 90.0% vs. 103.7%) [34]. This evidence concerns the gene AMH and ovarian hyperstimulation syndrome.