In our experiments, the most significant upregulated genes included FANCD2, a player in Fanconi anemia (FA) [57], and several genes playing a role in the HR-mediated repair of double-strand breaks (DSBs) such as BARD1, BRCA2, RAD51 and DNA2 [58,59]. The gene discussed is BRCA2; the disease is Fanconi anemia.