Oral administration of sodium propionate could ameliorate dextran sulfate sodium (DSS)-induced colitis in mice mainly by reducing cecal inflammation, including IL-1β, IL-6, and TNF-α mRNA expression, and oxidative stress [20], while inducing antimicrobial peptides [21], regenerating islet-derived protein type 3 [Reg3] lectins. This evidence concerns the gene IL1B and colitis.