While excessive inflammation appears to increase the likelihood of abnormal cortical evolution, cytokines involved in the pathogenesis of MIA and autism, including IL-6, IL-TNF-, IL-1, and IL-17, are less attentive to TNF-α and IL-6 and play a physiological role in the proliferation of neuronal cells, differentiation, and abidance [70,71]. The gene discussed is IL6; the disease is autism.