Given the excessive inflammatory response in SARS-CoV2 and CDI infections, with elevated levels of IL-6, TNF-α, IL-1β, and IP-10, as well as associated dysbiosis mentioned in the literature [3,4,48], FMT might be a key factor in CDI treatment or more severe clinical scenarios like SARS-CoV-2 and CDI co-infections. The gene discussed is CXCL10; the disease is coinfection.