After comparing the outcomes in the related literature with the findings in our results, it was shown that the high expression level of WT1, IGFBP1, and APOE, and the low expression level of JUN was verified by previous clinical cases or experiments on GC, and the down-regulations of CA10 and GRIA2 were demonstrated in other tumor origins, where the results were all matched with our calculations. The gene discussed is JUN; the disease is neoplasm.