ITGAM and neoplasm: Neutrophils have functional plasticity with either pro‐ or anti‐tumor affinity within the TIME.[38] In murine models, neutrophil polarization can contribute to either accelerating or halting tumor progression.[39] We demonstrate that CD40 treatment significantly increased neutrophil (Ly6G+Ly6C‐CD11b+) infiltration within tumor by on days 7 and 14 (Figure S5A, Supporting Information).