It has been confirmed that CDCP1 was overexpressed in bladder cancer cells, mediated by m6A methyltransferase METTL3, m6A reader YTHDF1 preferentially recognizes m6A residues on CPCP1 3′-UTR and enhances the CDCP1 translation, promotes bladder cancer tumorigenesis in vitro and in vivo [86]. Here, YTHDF1 is linked to urinary bladder cancer.