Of ACSLs, ACSL5 was considered as the target gene, although lysoPCs may stimulate ACSL isoform activations and promote isoform‐specific biological processes, for example, ACSL1‐involved pro‐inflammation, ACSL3‐regulated androgen responsiveness or ACSL4‐and ACSL5‐related tumour suppression.4 The gene discussed is ACSL4; the disease is neoplasm.