IL-10 can inhibit the secretion of IL-4 and IL-17, reduce the secretory activity of Th2 and Th17 cells, regulate the Th1/Th2 and Th17/Treg balance, effectively limit immune overreaction and inflammatory factors, maintain immune homeostasis, and reduce airway inflammation [33], as well as IFN-γ, IL-4, IL-17, and IL-10 as the functional cytokines of Th1, Th2, Th17, and Treg cells, which can reflect the inflammation in asthma [44]. The gene discussed is IL17A; the disease is asthma.