Decreased ABI1 expression was found in hematopoietic stem/progenitor cells in patients with myeloproliferative neoplasm (MPN), and murine bone marrow‐targeted depletion of ABI1 was shown to be associated with an MPN‐like phenotype mechanistically linked to activation of SFKs, STAT3, and NF‐κB pathways [24]. This evidence concerns the gene NFKB1 and myeloproliferative disorder.