Cancer cells can suppress apoptosis by the overexpression of anti-apoptotic proteins (e.g., B-cell lymphoma-2 (BCL-2), BCL-XL, and myeloid cell leukemia-1 (MCL-1)) to downregulate or inactivate pro-apoptotic proteins (BCL-2-associated X, BCL-2-associated K, and BCL-2-related ovarian killer)[59]. This evidence concerns the gene MCL1 and cancer.