In addition, HPV18 promoted YY1 expression by reducing the 3’ untranslated region of miR-29a targeted YY1 mRNA.[51] Endometriosis has been considered as a precursor to neoplasms associated with endometriosis.[52] In endometriosis, the PROMO and AnimalTFDB databases predict that lncRNAs (MALAT1, NEAT1, SNHG22, and XIST) may interact with YY1.[53]. Here, SNHG22 is linked to endometriosis.