PKMYT1 promotes the dryness of cancer stem cells through β-catenin ubiquitin degradation, leading to lung cancer progression.[17] In additional, LINC01089 inhibited by YY1 competes with endogenous RNA of miR-301b-3p to promote hydroxyprostaglandin dehydrogenase expression.[18]. Here, YY1 is linked to lung carcinoma.