During this process, overexpression of Oct4 and c-MYC can induce higher expression of enolase 1 (Eno1), heat shock protein 90 α family class b member 1 (HSP90AB1), eukaryotic elongation factor 2 (Eef2), vinculin (VCL), Trail, and p53 to suppress other breast cancer cells [184]. This evidence concerns the gene ENO1 and breast carcinoma.