When we tried to dissect the differential molecular mechanism underneath the low‐and high‐risk group, GSVA disclosed high activity of some pathways in the high‐risk group, which are known to facilitate tumor cell survival and growth, including hypoxia, DNA repair, glycolysis, EMT, MYC targets V1, MYC targets V2, MTORC1 signaling, DNA replication, and regulation of autophagy. This evidence concerns the gene MYC and neoplasm.