Kang et al. (25) revealed that QG inhibits the formation of macrophage-derived chemokine (MDC/CCL22) in the human keratinocytes (HaCaT) by mediating signal transducer and activator of transcription 1 (STAT1), suppressor of cytokine signaling 1 (SOCS1) and transforming the growth factor-β1 (TGF-β1); hence its potential as an immunotherapeutic agent against inflammatory diseases such as atopic dermatitis (AD). This evidence concerns the gene SOCS1 and atopic eczema.